our results suggest that attenuation of cardiac cell death by Glisodin treatment results in a significant prevention of the development of diabetic cardiomyopathy.
We have shown that a gliadin peptide fused to Cu–Zn SOD allows it to physically interact with a potential receptor on the surface of pithelial cells without altering the antioxidant activity of the SOD protein.
supplementation with an extract rich in SOD activity promoted antioxidant status and protected against increased inflammation in the serum of professional rowers but had no effect on oxidative damage induced by exhaustive exercise.
The purpose of this study was to examine changes in performance and metabolic parameters in collegiate soccer players during preseason preparation and to determine the impact of a nutraceutical blend proposed to reduce oxidative stress.
GliSODin could be considered a food supplement in the trials of minimizing oxidative stress disorders due to radiation exposure.
Orally supplemented GliSODin has been shown scientifically to offer a therapeutic means for the prevention and treatment
of many conditions associated with inflammation and oxidative stress
Antioxidants’ are all individual in their properties; in their cellular compartmentalisation, in their ability to cross cell membranes, in the speed with which they undergo chemical reactions (kinetics), in their solubility, in their bioavailability, in their interactions with other molecules and in their ability to act as messengers, sending signals which initiate critical processes.
These finding suggests that GliSODin prevents stress-induced impairment of cognitive function and maintains neurogenesis in the hippocampus through antioxidant activity.
Antioxidant supplementation resulted in an increase in SOD levels, confirming the oral bioavailability of the compound in FIV-infected cats.
The extreme toxicity of oxygen is related to its unique capacity of generating free radicals, which have long been recognized as harmful compounds on biological molecules.
